6 edition of Myocardial and Skeletal Muscle Bioenergetics found in the catalog.
June 30, 1986
Written in English
Advances in Experimental Medicine and Biology
|The Physical Object|
|Number of Pages||692|
Tissue-specific and substrate-specific mitochondrial bioenergetics in feline cardiac and skeletal Available via license: CC BY-NC-ND Content may be subject to copyright. In skeletal muscle the authors report a similar depression in mitochondrial transcription factors suggesting a common mechanism for both cardiac and skeletal muscle bioenergetic deficit in HF in animals. In animal models, the general conclusion is that PGC1α is upregulated during compensated hypertrophy and downregulated in by:
Recall that cardiac muscle shares a few characteristics with both skeletal muscle and smooth muscle, but it has some unique properties of its own. Not the least of these exceptional properties is its ability to initiate an electrical potential at a fixed rate that spreads rapidly from cell to . ORIGINAL RESEARCH Impaired cardiac and skeletal muscle bioenergetics in children, adolescents, and young adults with Barth syndrome Adil Bashir1,2, Kathryn L. Bohnert3, Dominic N. Reeds4, Linda R. Peterson4, Adam J. Bittel3, Lisa de las Fuentes4, Christina A. Pacak5, Barry J. Byrne5 & W. Todd Cade3,4 1 Department of Radiology, Washington University School of Medicine, St. Louis, MissouriCited by:
Arnold M. Katz, in Muscle, Skeletal, Cardiac, and Smooth Muscle. The heart has been known to be a muscle for at least 2, years. The Heart, a book of the Hippocratic corpus that is probably of Hellenistic origin, states: “The heart is a very strong muscle because of the firmness of its flesh” (64). The heart’s function as a. In cardiac, skeletal, and some smooth muscle tissue, contraction occurs through a phenomenon known as excitation contraction coupling (ECC). ECC describes the process of converting an electrical stimulus from the neurons into a mechanical response that facilitates muscle movement.
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Myocardial Acidosis and the Mitigation of Tissue ATP Depletion in Ischemic Cardiac Muscle: The Role of the Mitochondrial ATPase. Myocardial Acidosis and the Mitigation of Tissue ATP Depletion in Ischemic Cardiac Muscle: The Role of the Mitochondrial ATPase.
Pages Rouslin, William. Myocardial Acidosis and the Mitigation of Tissue ATP Depletion in Ischemic Cardiac Muscle: The Role of the Mitochondrial ATPase.- Vascular Metabolism and Energetics.- Myocardial Glutamate Dehydrogenase Activity Myocardial Preservation, Ischemia: Cellular Mechanisms --Compartmentation and Functional Mechanisms in Myocardial Failure and Myocardial Infarction --Myocardial Protection of Hypertrophied Hearts by Administration of Cardioplegia According to Regional Myocardial Temperature --Heart Myocytes Myocardial and Skeletal Muscle Bioenergetics book Models of the Cellular Response to Ischemia --Biologic Basis for Limitation of.
Cardiac muscle contains a great many mitochondria, which produce ATP for energy. This helps the heart resist fatigue. Contractions of cardiac muscle are involuntary, like those of smooth muscle.
Cardiac muscle, like skeletal muscle, is arranged in bundles, so it appears striated, or striped. Full text Full text is available as a scanned copy of the original print version. Get a printable copy (PDF file) of the complete article (K), or click on a page image below to browse page by page.
Effect of Calcium Antagonists on Vasopressin Induced Changes in Myocardial and Renal Pyridine Nucleotides in the Intact Rat.- The Effect of Creatine Analogue Substitution on the Post Tetanic Response of Fast Muscle.- V.
Divalent Ions and Cellular Bioenergetics.- Calcium Compartmentation in Mammalian Myocardium A valuable study of the science behind the medicine, Muscle: Fundamental Biology and Mechanisms of Disease brings together key leaders in muscle biology. These experts provide state-of-the-art insights into the three forms of muscle--cardiac, skeletal, and smooth--from molecular anatomy, basic physiology, disease mechanisms, and targets of therapy.
Paradigms for understanding muscle bioenergetics then can be understood from a set of basic premises of biochemical energy balance 1) ATP provides the energy for all forms of muscle work; 2) chemical energy is stored in cells as phosphocreatine, a biochemical capacitor; 3) the sum of the coupled ATPases sets the demand side of the balance and Cited by: 6.
These experts provide state-of-the-art insights into the three forms of muscle--cardiac, skeletal, and smooth--from molecular anatomy, basic physiology, disease mechanisms, and targets of therapy.
Commonalities and contrasts among these three tissue types are highlighted. McDaniel H.G., Jenkins R.L. () Myocardial Glutamate Dehydrogenase Activity. In: Brautbar N. (eds) Myocardial and Skeletal Muscle Bioenergetics.
Advances in Cited by: 1. In myocardial infarction, due to regional ischemia both cause and effect are more easily defined. It is the purpose of this report to describe changes in compartmentation and specific mechanism in two conditions: myocardial failure and myocardial by: 1.
In fact, the first reports describing the biochemical adaptations of skeletal myocytes in response to exercise were published over 60 years ago.
The earliest investigations were descriptive and chronicled the exercise-induced increases in the abundance of key bioenergetic enzymes in both cardiac and skeletal muscles [18,21,22].
In the decades Author: Scott K. Powers, Matthew Bomkamp, Mustafa Ozdemir, Hayden Hyatt. Introduction. The heart and skeletal muscles undergo dramatic changes with aging that contribute to reduce exercise capacity as we age.
In the context of the heart amongst the most important changes is a progressive cardiomyocyte loss and replacement fibrosis; factors that are key to the impaired diastolic function and increased risk of heart failure seen with by: Barth syndrome (BTHS) is an X‐linked condition characterized by altered cardiolipin metabolism and cardioskeletal myopathy.
We sought to compare cardiac and skeletal muscle bioenergetics in children, adolescents, and young adults with BTHS and unaffected controls and examine their relationships with cardiac function and exercise by: Abstract.
A number of reports have now appeared demonstrating a marked inhibition of the mitochondrial oligomycin-sensitive ATPase as the result of the interruption of blood flow (ischemia) in a number of tissues including liver, 1,2 kidney, 3,4 skeletal muscle 5 and cardiac muscle. 6–9 By and large, the ATPase inhibition observed in ischemic tissues appears to be reversible if blood flow is Cited by: 5.
Elucidation of the mitochondrial regulatory mechanisms for the understanding of muscle bioenergetics and the role of mitochondria is a fundamental problem in cellular physiology and pathophysiology.
The cytoskeleton (microtubules, intermediate filaments, microfilaments) plays a central role in the maintenance of mitochondrial shape, location, and : Andrey V. Kuznetsov, Sabzali Javadov, Michael Grimm, Raimund Margreiter, Michael J. Ausserlechner, J. The metabolism and performance of myocardial and skeletal muscle are impaired in heart failure (HF) patients.
Exercise training improves the performance and benefits the quality of life in HF patients. The purpose of the present study was to determine the metabolic profiles in myocardial and skeletal muscle in HF and exercise training using MRS, and thus to identify targets for clinical MRS in Author: Mingshu Shi, Øyvind Ellingsen, Tone Frost Bathen, Morten A.
Høydal, Tomas Stølen, Morteza Esmaeili. This study provides evidence of the impaired myocardial bioenergetics in experimental uremia and that skeletal muscle bioenergetics changes mirror those seen in the cardiac tissue. Therefore, skeletal muscle could potentially be used as a surrogate tissue during clinical cardiac NMR by: 1.
Isolated rat cardiac and skeletal muscle mitochondria readily oxidize lactate and pyruvate in vitro, and the rate of lactate oxidation is ∼10% greater than that of pyruvate. Oxidation of both lactate and pyruvate by muscle mitochondria is blocked by the MCT inhibitor cinnamate, whereas the LDH inhibitor oxamate blocks lactate, but not.
Furthermore, we have previously demonstrated that glycerintreated fiber of cardiac muscle was far less sensitive to changes of Ca" concentration than those of skeletal muscle . These findings correlate well with the difference of Ca2''-sensitivity between skeletal and cardiac natural actomyosin as measured by the superprecipitation (Table I).Cited by: 2.Developmental, physiological, and evolutionary origins of variation in skeletal muscle bioenergetics | Find, read and cite all the research you need on ResearchGate Book.
Myocardial and.Cardiac and skeletal muscle energy metabolism. It has long been known that the energetic characteristics of muscle cells are not uniform but adapted to the specific requirements of a given muscle.
Schematically, two extreme metabolic patterns have been by: